King Cobra Venom
King cobra venom contains α-neurotoxins (NTXs) in addition to phospholipase A2 (PLA2) and cardiotoxin (CTX), and a single bite can deliver up to 400–500 mg of venom, about fifteen thousand times the LD50 dose for mice. The toxin affects the postsynaptic nicotinic acetylcholine receptors. The Toxin increases the threshold level which means it would require more excitatory signals to fire the neuron. Which means the toxin is inhibitory. After being bitten losing of motor and sensory function occurs which then could lead to paralysis and death.
Acetylcholine are normally excitatory enzyme that are found in the synapse, which passes on the signal from on neuron’s axon to another neuron’s dendrite by attaching themselves to the protein receptor located on the dendrite of the next neuron cell. The next neuron which the acetylcholine attached to will then fire once they reached the threshold potential.
Acetylcholine are normally excitatory enzyme that are found in the synapse, which passes on the signal from on neuron’s axon to another neuron’s dendrite by attaching themselves to the protein receptor located on the dendrite of the next neuron cell. The next neuron which the acetylcholine attached to will then fire once they reached the threshold potential.